Background: Subarachnoid hemorrhage most commonly occurs secondary to trauma, aneurysms, or aggressive tumors. Benign tumors such as vestibular schwannomas are an uncommon cause of subarachnoid hemorrhage. Here we present a case of acute neurologic decline secondary to subarachnoid hemorrhage arising from a benign vestibular schwannoma.

Methods: The methods in this study include case presentation with operative findings and literature review.

Results: A 39 year old male presented with acute hearing loss, severe immediate headache, dizziness, facial numbness, and meningismus. Non-contrast head Computed Tomography (CT) demonstrated subarachnoid hemorrhage. CT angiogram and magnetic resonance imaging revealed a vascular tumor of the cerebellopontine angle with widening of the internal auditory canal, consistent with a vestibular schwannoma. The tumor was approached by retrosigmoid transmeatal craniotomy with complete surgical resection. The pathology revealed vestibular schwannoma.

Conclusion: Although extremely uncommon, benign tumors such as vestibular schwannomas may have an unusual presentation such as spontaneous subarachnoid hemorrhage.

Background: The thalamic syndrome was described in 1906 by Déjerine and Roussy. It is characterized by transient hemiparesis, hemianaesthesia, allodynia, mild hemiataxia and severe pain on the affected side. The syndrome is related to lesions in the posterior nuclei of the thalamus, usually of vascular aetiology. The aim of this work is to report a patient with a central pain Déjerine-Roussy-like syndrome due to an ischemic lesion in the dorsal pons

Case report: A 69-year-old hypertense male was admitted in May 2015 with mild left hemiparesis and hemihypoesthesia. The brain Computed Tomography (CT) scan showed a small hypodense lesion in the tegmentum of the right hemipons. Seven months later, the patient was admitted again due to intense painful paresthesias on the left side of the body, including the face. A new brain CT scan showed the same pontine infarct. The patient was successfully treated with pregabalin (75 mg twice a day)

Discussion: The reported patient presented a Déjerine-Roussy like syndrome with a pontine topography of the vascular lesion. It is probable that the ischemic damage partially affected the medial lemniscus and the lateral spinothalamic tract. The diagnosis of the central neuropathic pain is fundamental to select the most adequate treatment.

Endometrial adenocarcinoma rarely metastasizes to the heart. Few reports in the literature have discussed such situations with this type of carcinoma. Most metastases to the heart originate from lung, breast, thyroid, or skin (melanoma). In most cases, only one heart cavity is involved. We report a rare case of a 66 year old woman with endometrial adenocarcinoma who has metastases to both the right and left ventricles

Fibrous dysplasia is a condition characterized by excessive proliferation of bone-forming mesenchymal cells. It can affect one bone (monostotic type) or multiple bones (polyostotic type). It is usually observed in adolescents and young adults and comprises 7% of benign bone tumors. The etiology is not clear but genetic predisposition is suspected. It has a predilection for long bones as well as the craniofacial skeleton. The maxilla is the most commonly affected facial bone, with facial asymmetry being the usual complaint. The diagnosis is based on radiological and histopathological examination. There are different treatment approaches including monitoring, medical treatment or surgery. A 45-year-old female reported with a complaint of painless swelling on the left side of maxilla since 1 year. A diffuse intraoral bony hard, non tender swelling was seen in the left maxilla involving the premolar-molar region. Plain film radiographs and Computed tomography revealed ground glass appearance of the left maxilla. The lesion was excised and on histopathology showed features of fibrous dysplasia. Very few cases of Fibrous dysplasia manifesting in the older and postmenopausal age group are reported in the literature. Once diagnosed, routine follow-up should be done on a yearly basis with x-ray examination

We are presenting the case of a 38-year-oldpatientwith no significant medical history. He used to work as a professional diver but currently only partakes in recreational diving activities. Thirteen years ago, the patient reported self-limited para-paresis (24 hours) after diving, which improved after administering nor-mobaric oxygen and corticosteroids. This process was diagnosed as probable neurological Decompression Sickness (DCS).